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Pretreatment with a stem-cell-activating protein significantly enhances healing in mice, Stanford researchers say. The approach could eventually help people going into surgery or combat heal better from injuries they sustain.

A common signaling pathway unites diverse fibrotic diseases in humans, Stanford researchers have found. An antibody called anti-CD47, which is being tested as an anti-cancer agent, reverses fibrosis in mice.

A regulatory protein actively blocks the expression of non-neuronal genes in nerve cells, according to new Stanford research. The finding suggests there are many master regulators to help cell types maintain their identities.

After three patients were blinded following a treatment marketed as a stem cell clinical trial, Stanford ophthalmologist Jeffrey Goldberg calls for increased patient education and regulation.

Antibodies against the CD47 “don’t eat me” signal were shown in mice to be a safe and effective way to target five kinds of pediatric brain tumors, according to Stanford researchers.

When the arts and humanities play a role in medicine, patients, researchers and doctors can benefit. The winter issue of Stanford Medicine magazine features articles on the intersection of medicine with the arts and humanities.

Alpha cells can convert to insulin-producing beta cells in mice when just two genes are blocked, a new Stanford study shows. A similar mechanism may occur in people with diabetes.

Stanford researchers have developed a test that may help screen for cardiotoxicity in new chemotherapy drugs.

Microfluidics, electronics and inkjet technology underlie a newly developed all-in-one biochip from Stanford that can analyze cells for research and clinical applications.

Growing organs from one species in the body of another may one day relieve transplant shortages. Now researchers show that islets from rat-grown mouse pancreases can reverse disease when transplanted into diabetic mice.