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  • Three elected to National Academy of Medicine

    Hongjie Dai, Julie Parsonnet and Joseph Wu are among the 90 regular members and 10 international members elected this year to the academy, which aims to provide independent, scientifically informed analysis and recommendations on health issues.

  • Cause of deadly neurological disease found

    A drug may help children with Pelizaeus-Merzbacher disease, a deadly neurological disorder, according to a study by researchers at Stanford, UCSF and Cambridge.

  • Grad students receive lab coats

    During a ceremony at the Li Ka Shing Center, first-year doctoral students marked the beginning of their graduate careers by donning white lab coats and taking an oath of integrity.

  • Brain tumors integrate in neural wiring

    Tumors called high-grade gliomas wire themselves into the healthy brain, receiving and interpreting electrical signals from normal neurons, a Stanford study has found.

  • Irving Weissman honored for stem cell, cancer work

    Weissman and Johns Hopkins’ Bert Vogelstein will share the 2019 Albany Medical Center Prize in Medicine and Biomedical Research for discoveries in stem cell and cancer biology.

  • Scientists boost neuron recovery in rats

    Stanford researchers blocked a molecule to help restore neurons in rats in which the flow of fresh blood to the brain was reduced. The approach could lead to new treatments for people who have suffered a stroke or cardiac arrest.

  • Endowed professorships announced

    Michele Barry, Andra Blomkalns and Jeffrey Goldberg have been appointed to endowed professorships.

  • Microbiome initiative launched

    The Stanford Microbiome Therapies Initiative is backed by gifts from Marc and Lynne Benioff and Mark and Debra Leslie and is focused on developing and testing new disease therapies.

  • New ‘don’t eat me’ signal discovered

    Cancer cells are known to protect themselves using proteins that tell immune cells not to attack them. Stanford researchers have discovered a new “don’t eat me” signal, and blocking it may make cancer cells vulnerable to attack by the immune system.

  • Possible drug target for cardiomyopathy

    Stanford researchers have uncovered how a genetic mutation contributes to a heart disease known as familial dilated cardiomyopathy. Existing drugs correct the defect in heart cells grown in a petri dish, suggesting a new therapeutic target.