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New research suggests that lung fibrosis develops when scar tissue cells escape immune surveillance, suggesting potential therapy.

Unregulated cell growth seems to be a driver behind the growth of atherosclerotic plaques, changing the traditional story of plaque formation. The rapid cell growth in the arterial wall is similar to pre-cancerous growth in other tissues.

The Stanford developmental biologist was honored for a lifetime of work on the Wnt signaling pathway, which plays an important role in normal development and in cancer.

Stanford Medicine professors Michele Barry, Howard Chang, Thomas Clandinin and Thomas Rando were among the 15 Stanford faculty members elected to the American Academy of Arts and Sciences.

The researchers also identified a molecular pathway involved in turning back the clock on the cells. Drugs that could manipulate the pathway might be an effective substitute for exercise, they suggest.

Old human cells can become more youthful by coaxing them to briefly express proteins used to make induced pluripotent cells, Stanford researchers and their colleagues have found. The finding may have implications for aging research.

Stanford scientists have identified molecular drivers that put the “pause” in “diapause,” a life stage of the African killifish that suspends its development as an embryo.

Stanford researchers have found that drug-coated nanoparticles limit the development of atherosclerosis in mice, without side effects.

Stanford data scientists have shown that figuring out a single number can help them find the most dangerous cancer cells.

A new finding by Stanford researchers represents a missing link between two worlds — that of dietary science, and that of molecular and cellular biology.