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A study spearheaded by Stanford researchers indicates that at least 1 in 5 hospitalized COVID-19 patients develops new antibodies that attack their own tissue within a week of admission.

Researchers have identified the mechanism of scar formation in skin and demonstrated in mice a way to make wounds heal with normal skin instead of scar tissue.

Scientists have identified a key factor in mental aging and shown that it might be prevented or reversed by fixing a glitch in the immune system’s front-line soldiers.

A Stanford Medicine team used human stem cells to assemble a working nerve circuit connecting brain tissue to muscle tissue. The research could enable scientists to better understand neurological disorders that affect movement.

A single protein is a master regulator of mouse muscle function during aging, a Stanford study finds. Blocking this protein increased muscle strength and endurance in old animals. It may play a role in age-related muscle weakening in humans.

A common genetic deletion boosts the risk for schizophrenia by 30-fold. Generating nerve cells from people with the deletion has showed Stanford researchers why.

In laboratory studies, Stanford School of Medicine researchers have found a way to regenerate the cartilage that eases movement between bones.

A Stanford study shows that in severely ill COVID-19 patients, “first-responder” immune cells, which should react immediately to signs of viruses or bacteria in the body, instead respond sluggishly.

The researchers also identified a molecular pathway involved in turning back the clock on the cells. Drugs that could manipulate the pathway might be an effective substitute for exercise, they suggest.

Old human cells can become more youthful by coaxing them to briefly express proteins used to make induced pluripotent cells, Stanford researchers and their colleagues have found. The finding may have implications for aging research.