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A single protein is a master regulator of mouse muscle function during aging, a Stanford study finds. Blocking this protein increased muscle strength and endurance in old animals. It may play a role in age-related muscle weakening in humans.

A common genetic deletion boosts the risk for schizophrenia by 30-fold. Generating nerve cells from people with the deletion has showed Stanford researchers why.

Motivated by the loss of a patient, a doctor leads a research effort to uncover the molecular mechanisms of hemochromatosis in the heart.

In laboratory studies, Stanford School of Medicine researchers have found a way to regenerate the cartilage that eases movement between bones.

A Stanford study shows that in severely ill COVID-19 patients, “first-responder” immune cells, which should react immediately to signs of viruses or bacteria in the body, instead respond sluggishly.

Researchers at the Stanford School of Medicine have discovered that cells infected by viruses or bacteria send out a “don’t eat me” signal to avoid attack by the body’s immune system.

New Stanford graduate Nico Poux, a former pediatric oncology patient at Lucile Packard Children’s Hospital Stanford, hopes to bring his experience with cancer to future work as a physician-scientist.

Unregulated cell growth seems to be a driver behind the growth of atherosclerotic plaques, changing the traditional story of plaque formation. The rapid cell growth in the arterial wall is similar to pre-cancerous growth in other tissues.

The Stanford developmental biologist was honored for a lifetime of work on the Wnt signaling pathway, which plays an important role in normal development and in cancer.

Stanford Medicine professors Michele Barry, Howard Chang, Thomas Clandinin and Thomas Rando were among the 15 Stanford faculty members elected to the American Academy of Arts and Sciences.