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Stanford researchers found that activating bone stem cells helps repair fractures in diabetic mice. Applying a protein to the fracture site increased the expression of key signaling proteins and enhanced healing in the animals.

The California Institute for Regenerative Medicine awarded Samuel Strober, MD, $6.6 million to study a “deceptively simple” way to help kidney transplant recipients tolerate their new organ.

Stem cells produce a decoy protein to attenuate growth signals. Artificially regulating this pathway might help keep muscles supple in muscular dystrophy or during normal aging, researchers hope.

The work extends research by Stanford scientists who found that blocking CD47 might be useful in treating human cancer.

Using the CRISPR gene-editing technique in stem cells, Stanford researchers repaired the gene that causes sickle cell disease, and the mended stem cells were successfully transplanted into mice.

Mature fat cells produce a hormone that regulates the differentiation of nearby stem cells in response to glucocorticoid hormones and high-fat diets, Stanford researchers have found.

A new study shows that a diet deficient in valine effectively depleted the blood stem cells in mice and made it possible to perform a blood stem cell transplantation on them.

Making cell- or virus-based therapies for use in humans requires a rigid set of quality-control standards outlined by the Food and Drug Administration. A new Stanford facility will allow promising new therapies to be tested in the clinic.

Scientists have devised a way to destroy blood stem cells in mice without using chemotherapy or radiotherapy, both of which have toxic side effects.

A biological drug could be used to combat cardiovascular disease by targeting not mere risk factors such as high cholesterol or high blood pressure, but the actual lesions bearing direct responsibility: atherosclerotic plaques.