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Stanford scientists will direct a multidisciplinary, multi-institutional team focused on understanding in detail how tiny mutations in a protein, myosin, can cause the classic features of cardiomyopathy.

E-cigarette flavorings damage human blood vessel cells grown in the lab even in the absence of nicotine, Stanford researchers and their colleagues found. Cinnamon and menthol flavors were particularly harmful.

Stanford scientists have discovered a molecule that promotes the growth of collateral arteries in mice. The finding could open the door to developing therapies that help heal heart tissues damaged by disease or heart attack in humans.

Patients with cardiomyopathy have abnormally short telomeres in the cells responsible for heart contraction, Stanford researchers find. This disease hallmark opens new pathways for drug discovery.

Stanford researchers used genetic-editing tools and stem cell technology to uncover whether a genetic mutation linked to a heart rhythm disorder was benign or pathogenic.

Elevated maternal blood sugar when the fetal heart is forming has been linked to a heightened risk for congenital heart defects, according to a new Stanford study.

Stanford researchers have developed a test that may help screen for cardiotoxicity in new chemotherapy drugs.

Heart muscle cells made from induced pluripotent stem cells share gene expression patterns with native donor tissue, researchers discovered. These cells can be used to indicate people who should avoid certain medications that could damage their hearts.

A biological drug could be used to combat cardiovascular disease by targeting not mere risk factors such as high cholesterol or high blood pressure, but the actual lesions bearing direct responsibility: atherosclerotic plaques.

Doxorubicin is a chemotherapy drug used to treat many cancers, but it causes serious heart damage in some patients. Heart muscle cells made from the skin cells of breast cancer patients can be used to study this phenomenon.