Dr. Dewan was born and raised in Kalamazoo, Michigan. She attended college and medical school in Chicago at Northwestern University as part of the Honors Program in Medical Education (HPME). During her residency in Otolaryngology-Head & Neck Surgery Dr. Dewan trained in both Houston and Memphis. She completed a Laryngology fellowship at UCLA under Dr. Gerald Berke and Dr. Dinesh Chhetri.

As a Laryngologist, Dr. Dewan specializes in adult surgeries for swallowing, speaking and breathing disorders. She has a special interest in the surgical management of dysphagia, rehabilitation after total laryngectomy and the treatment of chronic cough. She has authored book chapters and published papers and continues to conduct clinical research in these areas, particularly with a focus on swallowing function and quality of life. She serves as a peer reviewer for multiple journals and a committee member within the American Academy of Otolaryngology - Head and Neck Surgery.

Clinical Focus

  • Otolaryngology
  • Laryngology

Academic Appointments

  • Assistant Professor - Med Center Line, Otolaryngology - Head & Neck Surgery Divisions

Honors & Awards

  • Travel Grant, National Spasmodic Dysphonia Association (2016)
  • Graduate with Distinction in Clinical Research, Feinberg School of Medicine (2009)
  • Honors in Otolaryngology, Feinberg School of Medicine (2008)
  • Medical Student Research Paper Prize, American Academy of Otolaryngology, Head and Neck Surgery (AAO-HNS) (2008)
  • Doris Duke Clinical Research Fellowship, Washington University in St. Louis (2007-2008)
  • Medical Student Summer Research Grant, Feinberg School of Medicine (2005)

Boards, Advisory Committees, Professional Organizations

  • Resident Member, American Broncho-Esophageal Association (2017 - Present)
  • Member, Laryngology and Bronchoesophagology Education Committee. American Academy of Otolaryngology - Head and Neck Surgery (2016 - Present)
  • Physician Advisor, Surgical Technology Training Program. Concorde Career College (2013 - 2015)
  • Peer Reviewer, JAMA Otolaryngology - Head and Neck Surgery (2012 - Present)
  • Peer Reviewer, The Laryngoscope (2012 - Present)
  • Resident Member, Sleep Disorders Committee. American Academy of Otolaryngology (2011 - 2015)
  • Resident Member, Allergy Immunology Review Committee. The Baylor College of Medicine (2011 - 2012)
  • Student Representative, Feinberg School of Medicine Curriculum Committee (2005 - 2009)

Professional Education

  • Board Certification: Otolaryngology, American Board of Otolaryngology (2016)
  • Board Certification, American Board of Otolaryngology, Otolaryngology (2016)
  • Fellowship:University of California Los Angeles School of Medicine (2017) CA
  • Residency:University of Tennessee (2015) TN
  • Residency:Baylor College of Medicine GME Office (2012) TX
  • Medical Education:Northwestern University Medical School (2009) IL
  • Fellowship, Washington University in St Louis, Doris Duke Clinical Research Fellowship (2008)


All Publications

  • Neuromuscular compensation mechanisms in vocal fold paralysis and paresis. Laryngoscope Dewan, K., Vahabzadeh-Hagh, A., Soofer, D., Chhetri, D. K. 2017


    Vocal fold paresis and paralysis are common conditions. Treatment options include augmentation laryngoplasty and voice therapy. The optimal management for this condition is unclear. The objective of this study was to assess possible neuromuscular compensation mechanisms that could potentially be used in the treatment of vocal fold paresis and paralysis.In vivo canine model.In an in vivo canine model, we examined three conditions: 1) unilateral right recurrent laryngeal nerve (RLN) paresis and paralysis, 2) unilateral superior laryngeal nerve (SLN) paralysis, and 3) unilateral vagal nerve paresis and paralysis. Phonatory acoustics and aerodynamics were measured in each of these conditions. Effective compensation was defined as improved acoustic and aerodynamic profile.The most effective compensation for all conditions was increasing RLN activation and decreasing glottal gap. Increasing RLN activation increased the percentage of possible phonatory conditions that achieved phonation onset. SLN activation generally led to decreased number of total phonation onset conditions within each category. Differential effects of SLN (cricothyroid [CT] muscle) activation were seen. Ipsilateral SLN activation could compensate for RLN paralysis; normal CT compensated well in unilateral SLN paralysis; and in vagal paresis/paralysis, contralateral SLN and RLN displayed antagonistic relationships.Methods to improve glottal closure should be the primary treatment for large glottal gaps. Neuromuscular compensation is possible for paresis. This study provides insights into possible compensatory mechanisms in vocal fold paresis and paralysis.NA Laryngoscope, 127:1633-1638, 2017.

    View details for DOI 10.1002/lary.26409

    View details for PubMedID 28059441

  • Pulsed dye laser treatment of primary cryptococcal laryngitis: A novel approach to an uncommon disease AMERICAN JOURNAL OF OTOLARYNGOLOGY Ihenachor, E. J., Dewan, K., Chhetri, D. 2016; 37 (6): 572-574


    An 82-year-old supplemental oxygen dependent woman with severe COPD presented with an eight month history of worsening hoarseness and stridor. Office laryngoscopy revealed laryngeal edema and ulcerative masses throughout the larynx. In-office biopsies were positive for Cryptococcus neoformans. This report details a novel approach to the treatment of cryptococcal laryngitis, a combination of in-office pulsed-dye laser (PDL) ablation and medical therapy. Despite treatment with oral fluconazole, the recommended treatment for cryptococcal laryngitis the patient continued to be symptomatic with dysphonia and throat discomfort. Repeated laryngeal exam demonstrated persistent cryptococcal nodules. The patient was subsequently effectively treated with an in-office PDL laser. This case demonstrates the efficacy of in-office laser treatment for residual laryngeal Cryptococcus. For patients like this one, who have failed medical therapy and are unfit for general anesthetic, the in-office laser provides an excellent alternative treatment approach.

    View details for DOI 10.1016/j.amjoto.2016.08.007

    View details for Web of Science ID 000387736600019

    View details for PubMedID 27654751

  • Incidence and implication of vocal fold paresis following neonatal cardiac surgery LARYNGOSCOPE Dewan, K., Cephus, C., Owczarzak, V., Ocampo, E. 2012; 122 (12): 2781-2785


    To study the incidence and implications of vocal fold paresis (VFP) following congenital neonatal cardiac surgery.Retrospective chart review.All neonates who underwent median sternotomy for cardiac surgery from May 2007 to May 2008 were evaluated. Flexible laryngoscopy was performed to evaluate vocal fold function after extubation. Swallow evaluation and a modified barium swallow study were performed prior to initiating oral feeding if the initial screening was abnormal.A total of 101 neonates underwent cardiac surgery during the study period. Ninety-four patients underwent a median sternotomy, and 76 of these were included in the study. Fifteen (19.7%) had vocal fold paresis (VFP) postoperatively. Almost 27% of the patients with aortic arch surgery had VFP while only 4.1% of the patients with nonaortic arch surgery developed VFP (P=0.02) Those patients who underwent aortic arch surgery weighed significantly less (P<0.01). All the patients with VFP had significant morbidity related to swallowing and nutrition (P=0.01) and required longer postsurgical hospitalization (P=0.02).The reported incidence of VFP following cardiac surgery via median sternotomy ranges between 1.7% and 67% depending on the type of surgery and the weight of the infant at the time of surgery. In our cohort, 19.7% had VFP. Surgery requiring aortic arch manipulation had a higher incidence of complications and required longer hospitalizations. These results may be used to improve informed consent and to manage postoperative expectations by identifying patients who are at higher risk for complications.

    View details for DOI 10.1002/lary.23575

    View details for Web of Science ID 000312540000029

    View details for PubMedID 22952115

  • Radiology Quiz Case 2 Nontraumatic atlantoaxial subluxation (also known as Grisel syndrome) ARCHIVES OF OTOLARYNGOLOGY-HEAD & NECK SURGERY Dewan, K., Giannoni, C. 2012; 138 (2): 199-201

    View details for Web of Science ID 000300525900016

    View details for PubMedID 22351870

  • Light-Induced Changes of the Circadian Clock of Humans: Increasing Duration is More Effective than Increasing Light Intensity SLEEP Dewan, K., Benloucif, S., Reid, K., Wolfe, L. F., Zee, P. C. 2011; 34 (5): 593-599


    To evaluate the effect of increasing the intensity and/or duration of exposure on light-induced changes in the timing of the circadian clock of humans.Multifactorial randomized controlled trial, between and within subject designGeneral Clinical Research Center (GCRC) of an academic medical center56 healthy young subjects (20-40 years of age)Research subjects were admitted for 2 independent stays of 4 nights/3 days for treatment with bright or dim-light (randomized order) at a time known to induce phase delays in circadian timing. The intensity and duration of the bright light were determined by random assignment to one of 9 treatment conditions (duration of 1, 2, or 3 hours at 2000, 4000, or 8000 lux).Treatment-induced changes in the dim light melatonin onset (DLMO) and dim light melatonin offset (DLMOff) were measured from blood samples collected every 20-30 min throughout baseline and post-treatment nights. Comparison by multi-factor analysis of variance (ANOVA) of light-induced changes in the time of the circadian melatonin rhythm for the 9 conditions revealed that changing the duration of the light exposure from 1 to 3 h increased the magnitude of light-induced delays. In contrast, increasing from moderate (2,000 lux) to high (8,000 lux) intensity light did not alter the magnitude of phase delays of the circadian melatonin rhythm.Results from the present study suggest that for phototherapy of circadian rhythm sleep disorders in humans, a longer period of moderate intensity light may be more effective than a shorter exposure period of high intensity light.

    View details for Web of Science ID 000291145800009

    View details for PubMedID 21532952

    View details for PubMedCentralID PMC3079938

  • Assessment of self-selection bias in a pediatric unilateral hearing loss study OTOLARYNGOLOGY-HEAD AND NECK SURGERY Lieu, J. E., Dewan, K. 2010; 142 (3): 427-433


    To examine the differences between participants and nonparticipants in a study of children with unilateral hearing loss that might contribute to selection bias.Case-control study.Academic pediatric otolaryngology practice.Comparison of clinical and sociodemographic characteristics between the 81 participants and 78 nonparticipants with unilateral hearing loss in a case-control study.Compared with nonparticipants, the study participants were younger but were diagnosed at an older age. Participants were more likely to have been diagnosed through a primary care screen and have normal ear anatomy, and less likely to have an attributed etiology for their unilateral hearing loss or tried assistive hearing devices. No other significant demographic, socioeconomic, or clinical differences were identified.Self-selection bias may jeopardize both internal and external validity of study results and should be evaluated whenever possible. Methods to minimize self-selection bias should be considered and implemented during the planning stages of clinical studies.

    View details for DOI 10.1016/j.otohns.2009.11.035

    View details for Web of Science ID 000276574600024

    View details for PubMedID 20172393

    View details for PubMedCentralID PMC2975441

  • Enlarged vestibular aqueduct in pediatric sensorineural hearing loss OTOLARYNGOLOGY-HEAD AND NECK SURGERY Dewan, K., Wippold, F. J., Lieu, J. E. 2009; 140 (4): 552-558


    Comparison of the Cincinnati criteria (midpoint >0.9 mm or operculum >1.9 mm) to the Valvassori criterion (midpoint > or =1.5 mm) for enlarged vestibular aqueduct (EVA) in pediatric cochlear implant patients.Cohort study.One hundred thirty pediatric cochlear implant recipients.We reviewed temporal bone CT scans to measure the vestibular aqueduct midpoint and opercular width.The Cincinnati criteria identified 44 percent of patients with EVA versus 16 percent with the Valvassori criterion (P < 0.01). Of those with EVA, 45 percent were unilateral and 55 percent were bilateral using Cincinnati criteria; 64 percent were unilateral and 36 percent bilateral using Valvassori criterion (P < 0.01). The Cincinnati criteria diagnosed 70 ears with EVA classified as normal using the Valvassori criterion (P < 0.01); 59 lacked another medical explanation for their hearing loss.The Cincinnati criteria identified a large percentage of pediatric cochlear implant patients with EVA who might otherwise have no known etiology for their deafness.

    View details for DOI 10.1016/j.otohns.2008.12.035

    View details for Web of Science ID 000264622700019

    View details for PubMedID 19328346

    View details for PubMedCentralID PMC2846828