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Dr. Haileselassie completed his medical degree at University of Missouri followed by residency at University of Washington. He attained clinical training and expertise in pediatric critical care medicine along with a master’s in health sciences from the Johns Hopkins University. After fellowship, Dr. Haileselassie attained postdoctoral T32 research training under Dr. Theodore Abraham at Johns Hopkins University, which focused on myocardial mechanics and redox biology. This was complimented by his Instructorship at Stanford University where he received further research training in mitochondrial biology under Dr. Mochly-Rosen in the Department of Chemical Systems Biology. Dr. Haileselassie was recruited to Stanford School of Medicine in the University Medical Line in 2020, and currently runs an NIH-funded translational research program aimed at understanding the role of alterations in mitochondrial dynamics in sepsis induced multiple organ dysfunction syndrome (MODS).
My laboratory is focused on understanding the cellular mechanisms which mediate end-organ failure in pediatric sepsis. Our current work focuses on determining the role of altered mitochondrial dynamics in sepsis-induced multi-organ dysfunction syndrome (MODS). Specifically, we focus on understanding the mechanisms that mediate derangements in mitochondrial fission and autophagy in sepsis.