Researchers have identified the mechanisms of scar formation in skin and demonstrated in mice a way to make wounds heal with normal skin instead of scar tissue.
A single protein is a master regulator of mouse muscle function during aging, a Stanford study finds. Blocking this protein increased muscle strength and endurance in old animals. It may play a role in age-related muscle weakening in humans.
In laboratory studies, Stanford School of Medicine researchers have found a way to regenerate the cartilage that eases movement between bones.
Researchers at the Stanford School of Medicine have discovered that cells infected by viruses or bacteria send out a “don’t eat me” signal to avoid attack by the body’s immune system.
New research suggests that lung fibrosis develops when scar tissue cells escape immune surveillance, suggesting potential therapy.
Video: Viral DNA is crucial to human development
REMS seminars will resume after summer