Dr. Baker is an Assistant Professor in the Division of Immunology and Rheumatology at Stanford University with a focus on patient care, bench research, and clinical trials. He studied at Harvard Medical School and trained in Internal Medicine at the Massachusetts General Hospital. He subsequently came to Stanford for his fellowship in Rheumatology, and spent several years in the laboratory of Dr. William Robinson conducting basic research related to characterizing novel autoantibodies in ANCA-vasculitis and investigating the immune profile of patients with IgG4-related disease. Under the mentorship of Dr. Mark Genovese, he has authored several investigator-initiated clinical trials with a focus on systemic lupus erythematosus, sarcoidosis, Sjogren's syndrome, and IgG4-related disease. He also serves as the Co-Director of the Stanford Multidisciplinary Sarcoidosis Program.

Clinical Focus

  • Rheumatology
  • IgG4-related disease
  • Sjogren's syndrome
  • Sarcoidosis
  • Cardiac sarcoidosis
  • Systemic lupus erythematosus
  • ANCA-vasculitis
  • Rheumatoid Arthritis
  • Osteoarthritis

Academic Appointments

Honors & Awards

  • Translational Research and Applied Medicine Award, Stanford University (9/1/18 - 9/1/19)
  • Scientist Development Award, Rheumatology Research Foundation (2/1/17 - 1/31/18)
  • KL2 Stanford Spectrum Mentored Career Development Award, National Institutes of Health (7/1/17 - 6/30/19)
  • Distinguished Fellow Award, American College of Rheumatology (11/28/16)
  • Bevra Hahn Distinguished Fellow Scholarship, California Rheumatology Alliance (5/24/16)
  • Ruth L. Kirschstein National Research Service Award, National Institutes of Health (7/1/16 - 1/31/17)
  • ACR/EULAR Exchange Program Award, American College of Rheumatology (6/12/17)

Professional Education

  • Master of Science, Stanford University, EPIDM-MS (2019)
  • Board Certification: Internal Medicine, American Board of Internal Medicine (2014)
  • Fellowship:Stanford University Rheumatology Fellowship (2016) CA
  • Board Certification: Rheumatology, American Board of Internal Medicine (2016)
  • Residency:Massachusetts General Hospital Internal Medicine Residency (2014) MA
  • Medical Education:Harvard Medical School (2011) MA
  • Bachelor of Arts, Pomona College (2004)

Research & Scholarship

Clinical Trials

  • A Crossover Study to Compare RAYOS to IR Prednisone to Improve Fatigue and Morning Symptoms for SLE Recruiting

    To compare the effect of RAYOS® versus immediate-release (IR) prednisone on fatigue as measured by Functional Assessment of Chronic Illness Therapy-Fatigue (FACIT-F).

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  • Safety and Efficacy of Filgotinib and Lanraplenib in Adults With Lupus Membranous Nephropathy (LMN) Recruiting

    The primary objective of this study is to evaluate the efficacy of filgotinib and lanreplenib (previously GS-9876) in adults with Lupus Membranous Nephropathy (LMN).

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  • Sarilumab in Patients With Glucocorticoid-Dependent Sarcoidosis Recruiting

    The purpose of this study is to compare the effectiveness and the safety of sarilumab in patients with glucocorticoid-dependent sarcoidosis.

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Lab Affiliations


All Publications

  • Dermatomyositis Associated With a Skull Base Chondrosarcoma JCR-JOURNAL OF CLINICAL RHEUMATOLOGY Baker, M. C., Smith, G. P., Miloslavsky, E. M. 2019; 25 (4): E50?E53
  • Security and Privacy Qualities of Medical Devices: An Analysis of FDA Postmarket Surveillance PLOS ONE Kramer, D. B., Baker, M., Ransford, B., Molina-Markham, A., Stewart, Q., Fu, K., Reynolds, M. R. 2012; 7 (7)


    Medical devices increasingly depend on computing functions such as wireless communication and Internet connectivity for software-based control of therapies and network-based transmission of patients' stored medical information. These computing capabilities introduce security and privacy risks, yet little is known about the prevalence of such risks within the clinical setting.We used three comprehensive, publicly available databases maintained by the Food and Drug Administration (FDA) to evaluate recalls and adverse events related to security and privacy risks of medical devices.Review of weekly enforcement reports identified 1,845 recalls; 605 (32.8%) of these included computers, 35 (1.9%) stored patient data, and 31 (1.7%) were capable of wireless communication. Searches of databases specific to recalls and adverse events identified only one event with a specific connection to security or privacy. Software-related recalls were relatively common, and most (81.8%) mentioned the possibility of upgrades, though only half of these provided specific instructions for the update mechanism.Our review of recalls and adverse events from federal government databases reveals sharp inconsistencies with databases at individual providers with respect to security and privacy risks. Recalls related to software may increase security risks because of unprotected update and correction mechanisms. To detect signals of security and privacy problems that adversely affect public health, federal postmarket surveillance strategies should rethink how to effectively and efficiently collect data on security and privacy problems in devices that increasingly depend on computing systems susceptible to malware.

    View details for DOI 10.1371/journal.pone.0040200

    View details for Web of Science ID 000306956300012

    View details for PubMedID 22829874

    View details for PubMedCentralID PMC3400651

  • Enhancement of DNA tumor vaccine efficacy by gene gun-mediated codelivery of threshold amounts of plasmid-encoded helper antigen BLOOD Leitner, W. W., Baker, M. C., Berenberg, T. L., Lu, M. C., Yannie, P. J., Udey, M. C. 2009; 113 (1): 37-45


    Nucleic acid-based vaccines are effective in infectious disease models but have yielded disappointing results in tumor models when tumor-associated self-antigens are used. Incorporation of helper epitopes from foreign antigens into tumor vaccines might enhance the immunogenicity of DNA vaccines without increasing toxicity. However, generation of fusion constructs encoding both tumor and helper antigens may be difficult, and resulting proteins have unpredictable physical and immunologic properties. Furthermore, simultaneous production of equal amounts of highly immunogenic helper and weakly immunogenic tumor antigens in situ could favor development of responses against the helper antigen rather than the antigen of interest. We assessed the ability of 2 helper antigens (beta-galactosidase or fragment C of tetanus toxin) encoded by one plasmid to augment responses to a self-antigen (lymphoma-associated T-cell receptor) encoded by a separate plasmid after codelivery into skin by gene gun. This approach allowed adjustment of the relative ratios of helper and tumor antigen plasmids to optimize helper effects. Incorporation of threshold (minimally immunogenic) amounts of helper antigen plasmid into a DNA vaccine regimen dramatically increased T cell-dependent protective immunity initiated by plasmid-encoded tumor-associated T-cell receptor antigen. This simple strategy can easily be incorporated into future vaccine trials in experimental animals and possibly in humans.

    View details for DOI 10.1182/blood-2008-01-136267

    View details for Web of Science ID 000262162800010

    View details for PubMedID 18832136

    View details for PubMedCentralID PMC2614641

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