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Professor Mehrdad Shamloo has held several positions at various biopharmaceutical companies in the San Francisco Bay Area, with demonstrated extensive focus on both CNS drug discovery and pre-clinical development. In 2008, Dr. Shamloo joined Stanford University and established the Behavioral and Functional Neuroscience Core Laboratory (BFNL), as well as his own research laboratory which focused on the furtherment of understanding of normal and pathological brain functions in neurological disorders such as Alzheimer’s disease (AD), Parkinson’s disease (PD), stroke and autism. Dr. Shamloo’s efforts and research are currently directed towards the neurodegenerative pathways responsible for Selective Neuronal Vulnerability of sensitive brain nuclei in the aforementioned disorders. Through these investigations, his aim is to understand the processes leading to the functional and behavioral malfunction in these neurological disorders, and to subsequently develop novel therapeutics to treat them. Long term, Dr. Shamloo seeks to accelerate the translation of experimental discoveries to novel therapeutic approaches, and to ultimately improve the quality of life for patients with brain disorders.Dr. Shamloo received his doctoral degree from the Wallenberg Neuroscience Center of Lund University in Sweden.
My laboratory aims to better understand normal and pathological brain function so that we can contribute to the discovery of novel therapeutic approaches for neurologic disorders such as Alzheimer’s disease (AD), Parkinson’s disease, stroke and autism. We have focused our efforts on targets involved in neurodegenerative pathways responsible for Selective Neuronal Vulnerability of sensitive nuclei in brain in these disorders. <br/><br/>We have identified Beta adrenergic system as one of key player in brain function in these disorders. Beta adrenergic modulation of the inflammation, cognition and pathological progression of CNS dieases are key projects in the lab. We are studying the mechansitic basis for modulation of inflammation, pathlogy and cognition in these disorsds.