Caleb Lareau

Instructor, Pathology

Bio

Profile moved to lab website: https://clareaulab.com/

Academic Appointments

Instructor, Pathology

Honors & Awards

30 Under 30- Science, Forbes (2022)
K99/R00 Pathway to Independence Award, National Human Genome Research Institute (2022)
STAT Wunderkind, STAT News (2022)
Parker Scholar, Parker Institute for Cancer Immunotherapy (2021)
Stanford Science Fellow, Stanford University (2020)
NIH Ruth L. Kirschstein National Research Service Award (F31), National Cancer Institute (2018)
NSF GRFP, National Science Foundation (2015)
DAAD Rise Fellow, Deutscher Akademischer Austauschdienst (2013, 2014)
Barry M. Goldwater Scholar, Goldwater Foundation (2013)

Professional Education

PhD, Harvard Medical School, Biological and Biomedical Sciences (2020)
Bachelor of Science, University of Tulsa, Biochemistry and Mathematics (2015)

2022-23 Courses

Cell, Gene, and Immune Therapies
BIOS 210 (Spr)

All Publications

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Anne Brunet

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Our lab studies the molecular basis of longevity. We are interested in the mechanism of action of known longevity genes, including FOXO and SIRT, in the mammalian nervous system. We are particularly interested in the role of these longevity genes in neural stem cells. We are also discovering novel genes and processes involved in aging using two short-lived model systems, the invertebrate C. elegans and an extremely short-lived vertebrate, the African killifish N. furzeri.

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Howard Y. Chang, MD, PhD

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Cancer > Cutaneous (Dermatologic) Oncology, Dermatology, General Dermatology
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Gerald Crabtree

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Jesse Engreitz

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Regulatory elements in the human genome harbor thousands of genetic risk variants for common diseases and could reveal targets for therapeutics — if only we could map the complex regulatory wiring that connects 2 million regulatory elements with 21,000 genes in thousands of cell types in the human body.

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Casey Gifford

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Kathleen Houlahan

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Chris C.S. Hsiung

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Selin Jessa

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Maya M. Kasowski

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Anshul Kundaje

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Jin Billy Li

Professor of Genetics
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The Li Lab is primarily interested in RNA editing mediated by ADAR enzymes. We co-discovered that the major function of RNA editing is to label endogenous dsRNAs as "self" to avoid being recognized as "non-self" by MDA5, a host innate immune dsRNA sensor, leading us to pursue therapeutic applications in cancer, autoimmune diseases, and viral infection. The other major direction of the lab is to develop technologies to harness endogenous ADAR enzymes for site-specific transcriptome engineering.

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