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Mackenzie Collins was the 500th pediatric patient to undergo a heart transplant at Stanford Medicine.

Hongjie Dai, Julie Parsonnet and Joseph Wu are among the 90 regular members and 10 international members elected this year to the academy, which aims to provide independent, scientifically informed analysis and recommendations on health issues.

Stanford scientists and their collaborators have discovered the protective role played by identity-morphing cells — and the gene behind them — in atherosclerosis, according to a new study.

Stanford researchers have uncovered how a genetic mutation contributes to a heart disease known as familial dilated cardiomyopathy. Existing drugs correct the defect in heart cells grown in a petri dish, suggesting a new therapeutic target.

A Stanford-led research team has mapped out a network of gene activity before and after heart failure to better understand how heart health declines.

Stanford scientists and their collaborators have devised an algorithm to predict the risk of a disease that, untreated, can lead to heart attack or stroke.

Stanford researchers have found a way to predict who will suffer heart problems from a common breast-cancer drug, as well as identified an FDA-approved medication that could mitigate those side effects.

Even a relatively simple heart defect makes a patient much more likely to develop cardiovascular disease as an adult, Stanford researchers say.

With no blood flow to his right lung, infant Carter Johnson was diagnosed with a rare condition called absent right pulmonary artery. His parents turned to Stanford Children’s Health for help.

Just 28 when she received a new heart at Stanford Hospital in 1991, Yolanda Ishaq went on to become the first heart transplant recipient to have a child at Stanford.