Nurses Return to Work After Approving Agreement

Physicians Unite on Need to Retain Welch Road Medical Offices

Vaccine Program Receives Federal Grant to Study Immune System Response to Viruses

Researchers Encourage Minority Patients to Participate in Cancer Studies

S.F. Opera Celebrities Perform for Palo Alto Fund-raiser

Center Party

Transplant Reunion

Volume 24No. 8 •AUGUST/SEPTEMBER 2000

The accomplishments of your colleagues and associates
are making a significant impact.
Detailed news releases and/or source material
are available at the
News Bureau of the Stanford University
Medical Center Office of News and Public Affairs,
701 Welch Road, Suite 2207, Palo Alto, CA 94304;
phone (650) 725-5376 or 723-6911;
and on the
World Wide Web
http://www-med.stanford.edu/center/communications/


EXERCISE - Published in the June issue of Health Psychology, a national study involving nearly 3,000 women 40 years of age and older finds that physical inactivity may be due, in part, to lack of contact with others who do exercise. Abby C. King, associate professor of health, research and policy, and Cynthia Castro, research associate, joined other colleagues nationally to publish the study - which also found that a majority of women would rather exercise on their own with instruction rather than with an instructor-led group.


SLEEP - Restless Leg Syndrome [RLS] may be much more common than previously recognized, stealing sleep from nearly 30 percent of the population. Clete Kushida, director of the Stanford Center for Human Sleep Research, and colleagues from Stanford and elsewhere reported at a meeting of the Associated Professional Sleep Societies in Las Vegas on June 14 that 368 out of 1,254 patients surveyed at a primary care clinic in Moscow, Idaho, showed symptoms of RLS.


ARTHRITIS GENE - Stanford scientists have found a mouse gene that transports pyrophosphate into and out of cells, and when that gene is defective, the animals have severe arthritis. Researchers, led by Stanford developmental biologist and Howard Hughes Medical Institute investigator David Kingsley, published their findings in the July 14 issue of Science.


-Mice with a defective gene develop bony deposits in their joints (bottom) typical of arthritis. Mice with the correct gene have normal joints (top).