Publications
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Immune activation in brain aging and neurodegeneration: too much or too little?
Lucin KM,
Wyss-Coray T.
Neuron.
2009;
64
(1):
110-22
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Stress hormones collaborate to induce lymphocyte apoptosis after high level spinal cord injury.
Lucin KM,
Sanders VM, Popovich PG.
J Neurochem.
2009;
110
(5):
1409-21
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The autophagy-related protein beclin 1 shows reduced expression in early Alzheimer disease and regulates amyloid beta accumulation in mice.
Pickford F,
Masliah E, Britschgi M, Lucin K, Narasimhan R, Jaeger PA, Small S, Spencer B, Rockenstein E, Levine B, Wyss-Coray T.
J Clin Invest.
2008;
118
(6):
2190-9
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Impaired antibody synthesis after spinal cord injury is level dependent and is due to sympathetic nervous system dysregulation.
Lucin KM,
Sanders VM, Jones TB, Malarkey WB, Popovich PG.
Exp Neurol.
2007;
207
(1):
75-84
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Mice lacking L1 cell adhesion molecule have deficits in locomotion and exhibit enhanced corticospinal tract sprouting following mild contusion injury to the spinal cord.
Jakeman LB,
Chen Y, Lucin KM, McTigue DM.
Eur J Neurosci.
2006;
23
(8):
1997-2011
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Spinal cord injury triggers systemic autoimmunity: evidence for chronic B lymphocyte activation and lupus-like autoantibody synthesis.
Ankeny DP,
Lucin KM, Sanders VM, McGaughy VM, Popovich PG.
J Neurochem.
2006;
99
(4):
1073-87
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