Publications
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A conserved acidic patch in the Myb domain is required for activation of an endogenous target gene and for chromatin binding.
Ko ER,
Ko D, Chen C, Lipsick JS.
Mol Cancer.
2008:
7
77
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Epigenetic regulation of gene expression by Drosophila Myb and E2F2-RBF via the Myb-MuvB/dREAM complex.
Wen H,
Andrejka L, Ashton J, Karess R, Lipsick JS.
Genes Dev.
2008;
22
(5):
601-14
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Loss of Drosophila Myb interrupts the progression of chromosome condensation.
Manak JR,
Wen H, Van T, Andrejka L, Lipsick JS.
Nat Cell Biol.
2007;
9
(5):
581-7
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v-Myb represses the transcription of Ets-2.
Wang DM,
Sevcikova S, Wen H, Roberts S, Lipsick JS.
Oncogene.
2007;
26
(8):
1238-44
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Myb proteins inhibit fibroblast transformation by v-Rel.
Fu SL,
Ganter B, Lipsick JS.
Mol Cancer.
2006:
5
54
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Functional evolution of the vertebrate Myb gene family: B-Myb, but neither A-Myb nor c-Myb, complements Drosophila Myb in hemocytes.
Davidson CJ,
Tirouvanziam R, Herzenberg LA, Lipsick JS.
Genetics.
2005;
169
(1):
215-29
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synMuv verite--Myb comes into focus.
Lipsick JS,
Genes Dev.
2004;
18
(23):
2837-44
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Role for a Drosophila Myb-containing protein complex in site-specific DNA replication.
Beall EL,
Manak JR, Zhou S, Bell M, Lipsick JS, Botchan MR.
Nature.
2002 Dec 19-26;
420
(6917):
833-7
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Mutation of the Drosophila homologue of the Myb protooncogene causes genomic instability.
Manak JR,
Mitiku N, Lipsick JS.
Proc Natl Acad Sci U S A.
2002;
99
(11):
7438-43
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Mutational analysis of the transcriptional activation domains of v-Myb.
Wang DM,
Lipsick JS.
Oncogene.
2002;
21
(10):
1611-5
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BS69, an adenovirus E1A-associated protein, inhibits the transcriptional activity of c-Myb.
Ladendorff NE,
Wu S, Lipsick JS.
Oncogene.
2001;
20
(1):
125-32
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BS69, an adenovirus E1A-associated protein binds to and inhibits the transcriptional activity of c-Myb
Lipsick, J.S,
Ladendorff, N.E., Wu, S.,.
Oncogene.
2001
20
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Transcriptional activation by the myb proteins requires a specific local promoter structure.
Ganter B,
Chao ST, Lipsick JS.
FEBS Lett.
1999;
460
(3):
401-10
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Functional analysis of carboxy-terminal deletion mutants of c-Myb.
Wang DM,
Dubendorff JW, Woo CH, Lipsick JS.
J Virol.
1999;
73
(7):
5875-86
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Transcriptional regulation by the carboxyl terminus of c-Myb depends upon both the Myb DNA-binding domain and the DNA recognition site.
Dubendorff JW,
Lipsick JS.
Oncogene.
1999;
18
(23):
3452-60
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Myb and oncogenesis.
Ganter B,
Lipsick JS.
Adv Cancer Res.
1999:
76
21-60
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Transformation by v-Myb.
Lipsick JS,
Wang DM.
Oncogene.
1999;
18
(19):
3047-55
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Myb-related Schizosaccharomyces pombe cdc5p is structurally and functionally conserved in eukaryotes.
Ohi R,
Feoktistova A, McCann S, Valentine V, Look AT, Lipsick JS, Gould KL.
Mol Cell Biol.
1998;
18
(7):
4097-108
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Overexpression of an alternatively spliced form of c-Myb results in increases in transactivation and transforms avian myelomonoblasts.
Woo CH,
Sopchak L, Lipsick JS.
J Virol.
1998;
72
(8):
6813-21
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D-type cyclins repress transcriptional activation by the v-Myb but not the c-Myb DNA-binding domain.
Ganter B,
Fu S, Lipsick JS.
EMBO J.
1998;
17
(1):
255-68
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Constitutive expression of full-length c-Myb transforms avian cells characteristic of both the monocytic and granulocytic lineages.
Fu SL,
Lipsick JS.
Cell Growth Differ.
1997;
8
(1):
35-45
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Myb binding sites within the N-ras promoter repress transcription.
Ganter B,
Lipsick JS.
Oncogene.
1997;
15
(2):
193-202
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Cells transformed by a v-Myb-estrogen receptor fusion differentiate into multinucleated giant cells.
Engelke U,
Wang DM, Lipsick JS.
J Virol.
1997;
71
(5):
3760-6
-
FAETL motif required for leukemic transformation by v-Myb.
Fu SL,
Lipsick JS.
J Virol.
1996;
70
(8):
5600-10
-
One billion years of Myb.
Lipsick JS,
Oncogene.
1996;
13
(2):
223-35
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Retinoic acid receptor alpha suppresses transformation by v-myb.
Smarda J,
Sugarman J, Glass C, Lipsick J.
Mol Cell Biol.
1995;
15
(5):
2474-81
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Dissociation of transcriptional activation and oncogenic transformation by v-Myb.
Chen RH,
Fields S, Lipsick JS.
Oncogene.
1995;
11
(9):
1771-9
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By-pass of TPA-induced differentiation and cell cycle arrest by the c-Myb DNA-binding domain.
Engelke U,
Smarda J, Lipsick JS.
Oncogene.
1995;
11
(4):
735-41
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Weak transcriptional activation is sufficient for transformation by v-Myb.
Engelke U,
Whittaker L, Lipsick JS.
Virology.
1995;
208
(2):
467-77
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Mutations in the DNA-binding and transcriptional activation domains of v-Myb cooperate in transformation.
Dini PW,
Eltman JT, Lipsick JS.
J Virol.
1995;
69
(4):
2515-24
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Transformation of myelomonocytic cells by the avian myeloblastosis virus is determined by the v-myb oncogene, not by the unique long terminal repeats of the virus.
Engelke U,
Lipsick JS.
J Virol.
1994;
68
(4):
2752-5
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c-Myb prevents TPA-induced differentiation and cell death in v-Myb transformed monoblasts.
Smarda J,
Lipsick JS.
Oncogene.
1994;
9
(1):
237-45
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c-Myb and v-Myb are differentially phosphorylated by p42mapk in vitro.
Aziz N,
Wu J, Dubendorff JW, Lipsick JS, Sturgill TW, Bender TP.
Oncogene.
1993;
8
(8):
2259-65
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Individual repeats of Drosophila Myb can function in transformation by v-Myb.
Bin X,
Lipsick JS.
J Virol.
1993;
67
(12):
7332-9
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Oncogenic truncation of the first repeat of c-Myb decreases DNA binding in vitro and in vivo.
Dini PW,
Lipsick JS.
Mol Cell Biol.
1993;
13
(12):
7334-48
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Dicistronic selection for nuclear proteins in living animal cells.
Smarda J,
Lipsick JS.
Gene.
1993;
137
(1):
145-9
-
Differential transcriptional activation by v-myb and c-myb in animal cells and Saccharomyces cerevisiae.
Chen RH,
Lipsick JS.
Mol Cell Biol.
1993;
13
(7):
4423-31
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Definition of functional domains in P135gag-myb-ets and p48v-myb proteins required to maintain the response of neuroretina cells to basic fibroblast growth factor.
Garrido C,
Leprince D, Lipsick JS, Stehelin D, Gospodarowicz D, Saule S.
J Virol.
1992;
66
(1):
160-6
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The Myb DNA-binding domain is highly conserved in Dictyostelium discoideum.
Stober-Grässer U,
Brydolf B, Bin X, Grässer F, Firtel RA, Lipsick JS.
Oncogene.
1992;
7
(3):
589-96
-
Expression of the CD4 gene requires a Myb transcription factor.
Siu G,
Wurster AL, Lipsick JS, Hedrick SM.
Mol Cell Biol.
1992;
12
(4):
1592-604
-
A highly conserved cysteine in the v-Myb DNA-binding domain is essential for transformation and transcriptional trans-activation.
Grässer FA,
LaMontagne K, Whittaker L, Stohr S, Lipsick JS.
Oncogene.
1992;
7
(5):
1005-9
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Protein truncation is not required for c-myb proto-oncogene activity in neuroretina cells.
Garrido C,
Grässer F, Lipsick JS, Stehelin D, Saule S.
J Virol.
1992;
66
(11):
6773-6
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Carboxy-terminal elements of c-Myb negatively regulate transcriptional activation in cis and in trans.
Dubendorff JW,
Whittaker LJ, Eltman JT, Lipsick JS.
Genes Dev.
1992;
6
(12B):
2524-35
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Protein truncation is required for the activation of the c-myb proto-oncogene.
Grässer FA,
Graf T, Lipsick JS.
Mol Cell Biol.
1991;
11
(8):
3987-96
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Determinants of sequence-specific DNA-binding by p48v-myb.
Garcia A,
LaMontagne K, Reavis D, Stober-Grässer U, Lipsick JS.
Oncogene.
1991;
6
(2):
265-73
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trans activation of gene expression by v-myb.
Ibanez CE,
Lipsick JS.
Mol Cell Biol.
1990;
10
(5):
2285-93
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Transformation by v-myb correlates with trans-activation of gene expression.
Lane T,
Ibanez C, Garcia A, Graf T, Lipsick J.
Mol Cell Biol.
1990;
10
(6):
2591-8
-
DNA-binding activity associated with the v-myb oncogene product is not sufficient for transformation.
Ibanez CE,
Garcia A, Stober-Grässer U, Lipsick JS.
J Virol.
1988;
62
(11):
4398-402
-
Specific amino acid substitutions are not required for transformation by v-myb of avian myeloblastosis virus.
Stober-Grässer U,
Lipsick JS.
J Virol.
1988;
62
(3):
1093-6
-
Structural and functional domains of the myb oncogene: requirements for nuclear transport, myeloid transformation, and colony formation.
Ibanez CE,
Lipsick JS.
J Virol.
1988;
62
(6):
1981-8
-
env-encoded residues are not required for transformation by p48v-myb.
Lipsick JS,
Ibanez CE.
J Virol.
1987;
61
(3):
933-6
-
c-myb protein expression is a late event during T-lymphocyte activation.
Lipsick JS,
Boyle WJ.
Mol Cell Biol.
1987;
7
(9):
3358-60
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v-myb does not prevent the expression of c-myb in avian erythroblasts.
Lipsick JS,
J Virol.
1987;
61
(10):
3284-7
-
The myb oncogene.
Lipsick JS,
Baluda MA.
Gene Amplif Anal.
1986:
4
73-98
-
Antibodies to the evolutionarily conserved amino-terminal region of the v-myb-encoded protein detect the c-myb protein in widely divergent metazoan species.
Boyle WJ,
Lipsick JS, Baluda MA.
Proc Natl Acad Sci U S A.
1986;
83
(13):
4685-9
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Expression of molecular clones of v-myb in avian and mammalian cells independently of transformation.
Lipsick JS,
Ibanez CE, Baluda MA.
J Virol.
1986;
59
(2):
267-75
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Cryptococcal myocarditis in acquired immune deficiency syndrome.
Lewis W,
Lipsick J, Cammarosano C.
Am J Cardiol.
1985;
55
(9):
1240
-
Biologically active proviral clone of myeloblastosis-associated virus type 1: implications for the genesis of avian myeloblastosis virus.
Perbal B,
Lipsick JS, Svoboda J, Silva RF, Baluda MA.
J Virol.
1985;
56
(1):
240-4
-
Avian myeloblastosis virus and E26 virus oncogene products are nuclear proteins.
Boyle WJ,
Lampert MA, Lipsick JS, Baluda MA.
Proc Natl Acad Sci U S A.
1984;
81
(14):
4265-9
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Identification of the leukemogenic protein of avian myeloblastosis virus and of its normal cellular homologue.
Boyle WJ,
Lipsick JS, Reddy EP, Baluda MA.
Proc Natl Acad Sci U S A.
1983;
80
(10):
2834-8
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