High-grade gliomas, a group of aggressive brain tumors, cease growing in mice if a signaling molecule called neuroligin-3 is absent or its activity is blocked with drugs, a Stanford team has shown.

Of the 37 million Americans who suffer from migraines, a few million progress to a chronic stage of having them more often than not. Stanford investigators hope to find out why.

Stanford researchers used advanced lab technologies to show, in mice, that symptoms of autism can be countered by reducing the ratio of excitatory to inhibitory neuronal firing in the forebrain.

A tiny set of nerve cells in a male mouse’s brain activates aggression. But a new Stanford study shows that the male’s susceptibility to this activation depends on whether it has been housed with other mice or in isolation.

Stanford researchers measured brain activity in PTSD patients before and after psychotherapy and found that they could predict how well patients would respond to treatment.

Lynn Koegel, who developed an early intervention for autism that taps children’s own motivations, began work at the School of Medicine and Lucile Packard Children’s Hospital this month.

Stanford Medicine is using a new software system that combines imaging from MRIs, CT scans and angiograms to create a three-dimensional model that physicians and patients can see and manipulate — just like a virtual reality game.

The brain hormone may help treat social impairments in children with autism whose baseline oxytocin levels are low before treatment, according to new Stanford findings.

The Stanford psychiatrist, neuroscientist and bioengineer will be honored for three distinct contributions to the medical field: optogenetics, hydrogel-tissue chemistry and research into depression.

Researchers at Stanford Medicine and the Gladstone Institutes will use the gift for gene editing and stem cell techniques to develop treatments for the neurodegenerative disorder.