Search Results
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Brain-to-muscle circuit in a dish
A Stanford Medicine team used human stem cells to assemble a working nerve circuit connecting brain tissue to muscle tissue. The research could enable scientists to better understand neurological disorders that affect movement.
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Molecule restores strength in old mice
A single protein is a master regulator of mouse muscle function during aging, a Stanford study finds. Blocking this protein increased muscle strength and endurance in old animals. It may play a role in age-related muscle weakening in humans.
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Neuronal abnormalities in schizophrenia
A common genetic deletion boosts the risk for schizophrenia by 30-fold. Generating nerve cells from people with the deletion has showed Stanford researchers why.
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Possible cure for iron-overload disease
Motivated by the loss of a patient, a doctor leads a research effort to uncover the molecular mechanisms of hemochromatosis in the heart.
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Method to regrow cartilage
In laboratory studies, Stanford School of Medicine researchers have found a way to regenerate the cartilage that eases movement between bones.
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Immune deviations seen in severe COVID-19 cases
A Stanford study shows that in severely ill COVID-19 patients, “first-responder” immune cells, which should react immediately to signs of viruses or bacteria in the body, instead respond sluggishly.
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How pathogens put the brakes on immune response
Researchers at the Stanford School of Medicine have discovered that cells infected by viruses or bacteria send out a “don’t eat me” signal to avoid attack by the body’s immune system.
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Cancer experience drives scientific curiosity
New Stanford graduate Nico Poux, a former pediatric oncology patient at Lucile Packard Children’s Hospital Stanford, hopes to bring his experience with cancer to future work as a physician-scientist.
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Potential treatment for lung fibrosis
New research suggests that lung fibrosis develops when scar tissue cells escape immune surveillance, suggesting potential therapy.
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Unregulated artery cell growth may drive atherosclerosis
Unregulated cell growth seems to be a driver behind the growth of atherosclerotic plaques, changing the traditional story of plaque formation. The rapid cell growth in the arterial wall is similar to pre-cancerous growth in other tissues.