Search Results

Beginning July 2023, Wu will lead the nation’s largest nonprofit organization dedicated to advancing cardiovascular health.

Motivated by the loss of a patient, a doctor leads a research effort to uncover the molecular mechanisms of hemochromatosis in the heart.

Unregulated cell growth seems to be a driver behind the growth of atherosclerotic plaques, changing the traditional story of plaque formation. The rapid cell growth in the arterial wall is similar to pre-cancerous growth in other tissues.

Stanford researchers have found that drug-coated nanoparticles limit the development of atherosclerosis in mice, without side effects.

Hongjie Dai, Julie Parsonnet and Joseph Wu are among the 90 regular members and 10 international members elected this year to the academy, which aims to provide independent, scientifically informed analysis and recommendations on health issues.

Stanford researchers have uncovered how a genetic mutation contributes to a heart disease known as familial dilated cardiomyopathy. Existing drugs correct the defect in heart cells grown in a petri dish, suggesting a new therapeutic target.

Stanford scientists will direct a multidisciplinary, multi-institutional team focused on understanding in detail how tiny mutations in a protein, myosin, can cause the classic features of cardiomyopathy.

E-cigarette flavorings damage human blood vessel cells grown in the lab even in the absence of nicotine, Stanford researchers and their colleagues found. Cinnamon and menthol flavors were particularly harmful.

Stanford researchers have found a way to predict who will suffer heart problems from a common breast-cancer drug, as well as identified an FDA-approved medication that could mitigate those side effects.

Stanford scientists have discovered a molecule that promotes the growth of collateral arteries in mice. The finding could open the door to developing therapies that help heal heart tissues damaged by disease or heart attack in humans.